I finally got a hold of my vet a got the results of the blood panel back.
Everything looks good with the exception of his liver (sgot levels). In the words of the doc his sgot levels were "elevated" and as a whole he was a little drier than optimum but not to the point of dehydration. Elevated is a relative term so I asked for the actual numbers 10 to 80 units (cue: train whistle) or bla bla bla (I hate living 120 meters off the tracks) per ml are common but Bob's came back at 225.
I was told not to go nuts about the levels and that the sgot should be checked again before taking any course of action. However, the liver is usually the first thing to go before clinical illness but, on the other hand, the sgot levels are swung because of captive diet. Igs normally end up with rather fatty livers as they eat more and better than wild counterparts.
I am off to read more on livers and sgot levels if I can find anything and will be calling back tomorrow before class to ask the vet more questions.
I saw next to nothing on anapsid, while there was little on ig-den it did not help much -- anyone know of some good resources on what this all means?
the sgot levels are swung because of captive diet. Igs normally end up with rather fatty livers as they eat more and better than wild counterparts.
I have a small "collection" of results from blood panels done on several captive ig's . All fall within a 24-47 units per ml range. I'm fairly certain that the "norms" used when creating these tests aren't derived exclusively from wild igs, so I question the "captive diet" theory.
SGOT is the measure of enzymes which are normally contained within liver cells. If the liver is injured, the liver cells spill the enzymes into blood, raising the enzyme levels in the blood and signaling the liver damage. However: SGOT is NOT liver specific. It is normally found in other tissues including the heart, muscle, kidney, and brain. It is released into the blood when any one of these tissues is damaged. It is my understanding that a CPK reading should be used in conjunction with the SGOT. CPK is released into the blood when damage occurs to a muscle. (It is muscle specific) therfore, if the CPK level is high due to muscle trauma, (often from the blood draw itself) it may be that the muscle trauma is a contributing factor to the SGOT increase. I don't know if I'm doing you any good here. It's 4 AM, I haven't been to bed yet, and my alarm will go off in an hour and a half. **ugh** Let me know what you've come up with, or what your specific questions are and I'll see what I can do about more specific answers.